5 grams of circulating glucose vs 10 grams (not total per day). I’m showing sucrose (which is 50% glucose / 50% fructose) because I didn’t have pure glucose. I used the term neurotoxin colloquially / incorrectly in relation to nerve damage caused by. diabetic neuropathy.
The problem with the “fake boogeyman” of white sugar is people often swap it out for honey, evaporated cane sugar, or some other sweetener that has a similar or stronger spike effect on your blood glucose. The poison is in the dose, glycemic load should be the first-order concern.
Learn more at: http://www.AnimatedDiabetesPatient.com
Diabetes is diagnosed through laboratory blood tests: the fasting blood glucose test, hemoglobin A1c test, and the oral glucose tolerance test. Serious complications arise with type 2 diabetes, if not diagnosed early, or if inadequately controlled. Regular check-ups and monitoring are stressed, as these can help delay or prevent many diabetes complications.
The mysterious onset of type 1 diabetes may have to do with what’s in your genes. Studies of twins show that if one twin has type 1 diabetes, the other has a 50 to 80 percent likelihood to also get the disease.
In the past 5 years, new technology that screens the whole genetic code allowed researchers to uncover at least 50 diabetes-associated genes. Interestingly, many of those genes also relate to other autoimmune diseases such as multiple sclerosis or celiac disease.
Stephan Kissler, Ph.D., Assistant Investigator in the Section on Immunobiology at Joslin Diabetes Center, studies those genes associated with multiple diseases using some of the newest lab techniques available. Video Rating: / 5
In observation of #WoundCareAwarenessMonth, infectious diseases specialist, Dr. Carlos Guerra, discusses diabetic wounds and prevention. Video Rating: / 5
What is diabetes mellitus? Diabetes mellitus is when there’s too much glucose, a type of sugar, in the blood. Diabetes mellitus can be split into type 1, type 2, as well as a couple other subtypes, including gestational diabetes and drug-induced diabetes. Find our full video library only on Osmosis: http://osms.it/more.
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(Visit: http://www.uctv.tv) Cristina Brickman, MD. Assistant Professor of Medicine, Division of Infectious Disease, UCSF. Recorded on 04/08/2017. Series: “UCSF Vascular Surgery Symposium” [6/2017] [Professional Medical Education] [Show ID: 32301]
Type 1 diabetes is a genetic, auto-immune disorder. You can not “catch” diabetes from anyone. Lol. 😛 Video Rating: / 5
Developed and produced for http://www.MDPracticeGuide.com, a CME resource for physicians and healthcare providers.
Animation Description: Under normal circumstances, bacterial infection results in the release of chemokines that attract circulating neutrophils to the endothelium. This process is known as chemotaxis.
A variety of molecules are expressed on the endothelial cell surface that allow the neutrophil to be captured, then roll along the endothelium, then adhere.
Following adherence, the neutrophil migrates into the subendothelial tissue to reach the site of infection.
The neutrophil engulfs the bacteria and eliminates them via breakdown within the phagosomes — a process known as phagocytosis.
In states of hyperglycemia, chemotaxis is reduced. Adherence is also adversely affected.
Phagocytosis is also impaired by hyperglycemia.
Hyperglycemia also adversely affects the macrophage system. Under normal circumstances, circulating monocytes are attracted to sites of infection, roll, adhere, and then migrate into the subendothelial space. The monocyte then transforms into a macrophage.
which is then activated by cytokines released by the bacteria. The activated macrophage then engulfs the bacteria.
However, hyperglycemia results in decreased activation of macrophages, thereby arresting the process of macrophage phagocytosis of bacteria.
In addition to affecting neutrophil and macrophage function, hyperglycemia also affects the complement cascade. Under situations of normal glycemia, bacteria can activate the complement cascade.
Activation of the complement cascade results in the formation of transmembrane protein channels known as membrane attack complex (MAC) in bacterial membrane.
Membrane attack complexes make the bacterial membrane porous and the rapid influx of fluid results in the bacterial cell death.
Hyperglycemia inhibits the proper activation of the complement cascade, thereby reducing another pathway of the immune system.
To learn more visit: http://www.AnimatedDiabetesPatient.com
Presently in medicine, diabetes is not a curable disease. In type 1 diabetes, patients need lifelong insulin treatment to keep their blood sugars in normal range to avoid complications. In type 2 diabetes, studies suggest that in the very early stages of the disease it can be reversed with a strict calorie diet. But once type 2 diabetes is well established, it cannot be completely cured. Type 2 diabetes patients need to keep their blood sugars in normal range by maintaining a balanced diet, exercising regularly, and taking their medications on a regular basis to avoid long-term complications.
